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This was evident by the inhibition of JNK, ERK and p38 phosphorylation and downregulation of cyt- C, Bax, cleaved CASP-3 and upregulation of BCL2 [139]

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271 Feeding endothelial cell-specific inactivated mice a high-calorie diet improved insulin resistance and decreased fat accumulation, 271 suggesting that the suppression of endothelial p53 could be a new target for the treatment of obese patients with metabolic abnormalities