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demonstrated that GLP-1 activates the cAMP/PKA signaling pathway by binding to its receptor GLP-1R and by then phosphorylating the PKA consensus site of NHE3 (101)

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Adds glucagon-receptor agonism on top of the dual-incretin mechanism, a contribution implicated in energy-expenditure and lipid-metabolism pathways in preclinical models

Nutritional approach to small intestinal bacterial overgrowth: A narrative review