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In contrast, metabolites such as 4-Aminobutanoate, 4-aminobutyraldehyde, 3-hydroxyanthranilate and porphobilinogen were elevated in the plasma of LC patients compared to the R group ( Figure 4C , Supplementary Figure 3B )

Current evidence suggests that inflammation contributes to EDC through upregulation of central inflammatory cytokines, overactivation of microglia and astrocytes, and hyperactivation of the HPA axis, leading to neurotransmitter imbalance, impaired synaptic plasticity, increased pyroptosis, and OSpositioning EDC as a prototypical inflammation-driven and stress-driven neuropsychiatric comorbidity (Xu et al., 2018

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