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Houtkooper, R.) 105138 (Sirtuins, 2016)

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When cells were treated with lysosomal reagents (e.g., ammonium chloride) or lysosomal protease inhibitors (e.g., chloroquine), the degradation of LC3-II was blocked, resulting in the accumulation of LC3-II

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Moulton, [email protected] This article was submitted to Autoimmune and Autoinflammatory Disorders, a section of the journal Frontiers in Immunology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers