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Hence, enterocyte DPP-4 is dispensable for the actions of sitagliptin to reduce TG excursion

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Ghrelin the endogenous ligand for GHS-R1a is known as the hunger hormone, and GHS-R1a agonism stimulates appetite through hypothalamic NPY/AgRP neuron activation

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However, evidence from human cell-based experiments suggests that these benefits are unlikely to result from direct actions on hepatocytes or HSCs, instead reflecting indirect metabolic and anti-inflammatory mechanisms

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