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However, while we and others previously showed that GIPR agonism remains fully efficacious to decrease body weight and food intake in mice deficient for Glp-1r 15,18 , here we found that loss of Glp-1r renders mice resistant to weight loss and inhibition of food intake by GIPR antagonism

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Research has documented Semaglutides neuroprotective effects in rodent neurodegeneration models , characterising reduced neuroinflammation, attenuated dopaminergic neuron loss in Parkinsons models, and addiction reward circuit modulation through central GLP-1R signalling

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