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Hepatic effects that are considered adverse include death, abnormal clinical chemistry parameters, obvious functional organ impairment, DNA and mitochondrial alteration, glutathione depletion, hepatocyte necrosis, enhanced replicative DNA synthesis, generation of reactive oxygen species, and increased peroxisome proliferation (in rodents) (Williams and Iatropoulos 2002)

Off-target activity may vary by concentration and species

Robino A, Concas MP, Catamo E, Gasparini P

foods that are high in sugar, carbs, and/or fat

( C , D ) Annexin V-FITC/PI apoptosis assays showing that FOXO4-DRI increases the apoptosis rate among senescent TM3 Leydig cells