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Studies suggest that the gut microbiome acts not merely as a passive entity but as an active biological influencer of GLP-1RA effectiveness, providing compelling support for microbiome-targeted precision therapies for conditions such as T2D and IBD [110,111]

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138 To date, there are limited data on clinically significant metabolic enzyme- or transporter-mediated DDIs involving GLP-1 RAs and a dual GLP-1/GIP RA