It is a bioactive form directly used by your cells, but has low stability in the digestive tract
These changes could be operational in neurodegenerative diseases, reflecting an imbalance in redox balance modulated by GSH
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An increasing body of preclinical and clinical evidence suggests that mitochondrial dysfunction, characterized by bioenergetic failure, heightened oxidative stress, dysregulated calcium handling, and impaired mitophagy, contributes to the etiopathogenesis of both CVDs and cognitive impairment