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Our data showing blunted responses to exogenous CCK and downregulated hypothalamic Cckbr receptor in Bbs5 / mice support a model in which impaired CCK signaling contributes to hyperphagia

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How does that make sense? The length of time she sees patients take the drugs varies, but some may have to take it indefinitely to suppress food cravings

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Endocrinology 141(3):12281235