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1993]Review Carnitine metabolism and human carnitine deficiency

W., Nizet, V

Our animal models, induced using endothelin-1 (ET-1), reflect clinically relevant cerebral I/R conditions yet may not fully capture the complexity of human pathophysiology, including inter-individual anatomical variation and comorbidities ( in vivo , including their potential off-target effects and clearance pathways

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Since different GSH-degrading enzymes are oriented to either intracellular or extracellular GSH pools, intracellular degrading enzymes directly decrease intracellular levels of GSH