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Under oxidative stress, glutathione reductase continuously regenerates it from its oxidized form (GSSG)

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The vitamin is then transferred to TC2, which serves as the circulating plasma transporter, hereditary defects in the production of TC2 and its receptor may produce functional deficiencies in B 12 and infantile megaloblastic anemia, and abnormal B 12 related biochemistry, even in some cases with normal blood B 12 levels

Nordlund, J

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Clin Chem 2005