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doi: 10.1111/j.1460-9568.2003.03033.x 161 SakuraiTAmemiyaAIshiiMMatsuzakiIChemelliRMTanakaHet al

[7] implicated 4-OH-E 2 in the induction of estrogen-responsive genes, a response that exhibited partial or no abrogation by coadministration with an antiestrogen, providing evidence for the ability of 4-OH-E 2 to carry out genetic upregulation via a pathway independent of ER signalling

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