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A better research question is which pathway, model or documentation standard fits the study objective
In this case, GSH levels are well below the normal concentration, and therefore there is no feedback inhibition of GCL
References Hutchison CA, Phillips S, Edgell MH, Gillam S, Jahnke P, Smith M
[7] Another aspect is that MB, in high IV doses, can paradoxically cause methemoglobinemia because it can auto-oxidize at excessive concentrations
1) from 3,4- cis -leucoanthocyanidins ( 2 ) to anthocyanidins ( 3 ) is thought to be catalysed by the Fe/-ketoglutarate-dependent dioxygenase leucoanthocyanidin dioxygenase (LDOX), also called anthocyanidin synthase (ANS) 8,9
As with any therapeutic option, patients are educated on proper use, potential side effects, and realistic outcomes to support safe and informed care
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