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In: Parnham MJ, Bruinvels J (eds) Discoveries in pharmacology, volume I: psycho- and neuro-pharmacology

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These collective findings support the view that PEM is not the result of a singular pathological process, but rather a dynamic network imbalance driven by the interplay of mitochondrial dysfunction, immune activation, and neuroinflammation, with distinct predominant mechanisms emerging across patient subtypes

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