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The exceptional in vitro potency is attributed to its mechanism of acting as an allosteric potentiator of HGF binding to c-Met (amplifying an endogenous signal), which produces non-linear dose-response curves

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In 216 patients with in-stent restenosis after coronary stent implantation who underwent optical coherence tomography of stented lesions, higher serum UA levels (7.0 mg/dL) were associated with a higher incidence of neoatherosclerosis (63% vs