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We discuss how copper also plays a key role in proteostasis and interacts either directly or indirectly with many of the key aggregate-prone proteins implicated in ALS, such as TDP-43, C9ORF72, SOD1 and FUS as well as the effect of their aggregation on copper homeostasis

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Intriguingly, EHMT2/G9a regulates intracellular innate immunity and macrophage polarization by modulating inflammatory genes such as Il6 and Il1b , and also influences NF-B and JAK/STAT signalling 40,41,42,43,44

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