Moreover, the data used for this model did not extend beyond 52 weeks, necessitating extrapolation
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It initiates a pulse of growth hormone release without significantly elevating secondary hormones like cortisol, prolactin, or aldosterone
Monica Boen
Diagnosis of LM LM presents clinically as a slow-growing, asymmetric macule with irregular, poorly defined borders and varied pigmentation (dark gray, light brown, dark brown, black, and white [areas of regression]).3,7 Albeit rarely, primary or recurrent LM can present clinically and histologically as an amelanotic lesion.8,9 Histology remains the diagnostic method of choice in LM.10 Characteristic histologic findings are the proliferation of atypical melanocytes along the dermal-epidermal junction
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