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Copper delivery to the CNS by CuATSM effectively treats motor neuron disease in SOD(G93A) mice co-expressing the copper-chaperone-for-SOD

Hemoglobin A1c levels tend to rise within weeks to months after discontinuation, returning toward pre-treatment values

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doi: 10.1007/s11010-009-0358-0

It is a close structural analogue of the naturallyoccurring GHK-Cu (Copper Tripeptide1), differing only in a single Nterminal residue: alanine in place of glycine (one methyl group)

In comparative studies with GHK-Cu, AHK-Cu has demonstrated distinct receptor interaction profiles, suggesting that subtle differences in amino acid sequence within copper tripeptides produce meaningfully different biological activity patterns