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Direct binding of eNOS to the scaffolding domain of Cav-1 is a well-accepted mechanism to repress eNOS activity that is associated with endothelial dysfunction and cardiovascular disease 39,40

Whilst some benefits have yet to produce scientific evidence, others are well documented

10.1016/j.bbi.2007.03.009 47

Cao et al., 2013)

2019 Aug 8;9(8):e025884

Cell viability assay A549 cells were plated into a 96-well plate and grown overnight, and then incubated with Bet v 1 or RSL-3 (HY-100218A, MCE, USA) at the indicated concentration in the presence or absence of 20 M Fer-1 (HY-100579, MCE, USA), 20 M DFO (HY-B0988, MCE, USA), 100 M Z-VAD-FMK (HY-16658B, MCE, USA), 100 M Nec-1 (T1847, Topscience, USA)