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In Ringer solution, although hemolysis was halved at 4% hematocrit when compared to 0.4% hematocrit, the difference was not significant

Because interaction data is sparse, underreported, and largely confined to preclinical rodent models

Autophagic flux tracing using tandem mRFP-GFP-LC3 probe showed that increase of autophagic flux caused by GSH depletion is augmented by rapamycin and attenuated by autophagic inhibitors 3-MA and Baf-A1 (Fig

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