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This can be achieved using biocompatible polymers, such as chitosan, polyethylene glycol (PEG), liposomes [61], and alginate

While repair and regeneration compose a topic in and of itself and worthy of another post, the point is that it would be desirable to seek new therapeutic agents that may interrupt the amplified oxidative/nitrosative cycle before necrosis is established in surviving hepatocytes so that they may recover, including through removal or repair of damaged mitochondria

Valproic acid aggravates epilepsy due to MELAS in a patient with an A3243G mutation of mitochondrial DNA

10.1007/s12325-012-0045-5 126 NasrN