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Accordingly, whereas PPAR agonists (fibrates) are classically assumed to improve lipid and cholesterol metabolism via their action on the liver, agonists at PPAR (thiazolidinediones) are assumed to improve insulin sensitivity by enhancing adipose tissue differentiation and fatty acid uptake 10,11,23

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Clinicians who broaden their diagnostic criteria and consider GLP1s within a liverfirst strategy may improve outcomes for patients who would otherwise go untreated or underdiagnosed

This is an evolving area, and expert groups recommend individual risk assessment rather than blanket fear

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