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Christoph Buettner, chief of the division of endocrinology at Rutgers Robert Wood Johnson Medical School, told Prevention that rapid adjustments in bodily systems have historically shown similar risks

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TNF is a crucial mediator of this pathology. TNF and another pro-inflammatory substance called interleukin 1, produce products like reactive oxygen species that damage both the filters and the elastic cells that wrap around their exterior

Concomitant medications like beta blockers blunt increases, while drugs that raise sympathetic tone can amplify them