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CRC-infiltrating Th17 cells induced the expression of G-CSF by CAF leading to the recruitment to the TME of CD11b + Gr1 + myeloid cells endowed with pro-angiogenic features, and thus promoting resistance to VEGF blockade therapy (137)

The column was kept at 30C and the flow rate was 0.5ml/min

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It binds mainly through van der Waals forces and hydrogen bonds, attaching to the isoalloxazine ring of the flavin adenine dinucleotide (FAD) domain in XO [1]

, is metabolized by the liver