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Since the GLP-1 receptor is expressed on iNKT cells, GLP-1 RAs could induce a dose-dependent inhibition of cytokine secretion by these cells, resulting in improved psoriatic disease [11, 12, 21, 22]

[3] Challenges in Establishing Optimal GLP-1 Dosing Regimens During the development phase, oral GLP-1 therapies can present particular challenges, however

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