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R.To EELuongR.LiongF.LiongS.OseghaleO.et al
doi: 10.3389/fonc.2022.998964 Received 20 July 2022 Accepted 24 August 2022 Published 23 September 2022 Volume 12 - 2022 Edited by Maria Rosaria De Miglio, University of Sassari, Italy Reviewed by Yusuke Yoshioka, Tokyo Medical University, Japan
Mechanistically, acetyl-CoA, a derivative of BCKAs, inhibits the interaction between PR domain-containing protein 16 (PRDM16) and peroxisome proliferator-activated receptor- (PPAR-) by acetylating the k915 site of PRDM16 to maintain WAT characteristics
doi:10.1111/cts.13109
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