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(June 2003)

To date, PARP inhibitors, including niraparib, rucaparib and olaparib have been approved by US-FDA to treat cancers, including prostate cancer, breast cancer and ovarian cancer, through disrupting DNA repair and replication pathways

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[12] These cellular effects may contribute to hypotheses about tissue-repair-associated pathways in preclinical models, but they do not establish human repair efficacy, clinical benefit, or safety

And a surprising number were simply normal adjustment periods misinterpreted as problems

However, in the initial cell density 5 10 3 cells, we observed lower cell death levels than 2.5 10 4