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Mitochondrial ROS in cardiac ischemia reperfusion and remodeling

Most studies discussed in this section were conducted in healthy individuals

For this role, SMAX1 D2 has been repurposed from an ATPase domain into a binding module through the loss of its nucleotide binding site and the use of its pore loop for allosteric stabilisation of the association between the F-box and the SL-bound receptor

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