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The structure consists of an intracellular segment, a transmembrane domain, and an extracellular segment

To test this model, we first generated RGKT deletion (RGKT) mutants in both full-length SMAX1 and the isolated SMAX1 D2 to mimic the original phenotypic d53 mutant reported by Zhou et al

Graphs were created using BioRender.com.

p53 deficiency decreases hepatic lipid peroxidation, inhibits the apoptosis of hepatocytes, and suppresses the development of nutritional steatohepatitis by suppressing the p66Shc signaling pathway