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Succinyladenosine accumulation has been reported in inborn errors of metabolism that disrupt purine metabolism and mitochondrial function, such as adenylosuccinate lyase (ADSL) deficiency and fumarase deficiency [49, 50], and its consistent upregulation in MMA may reflect secondary effects on nucleotide metabolism or mitochondrial energy balance, representing an underexplored aspect of MMA pathophysiology

The only theoretical and clinically proven supplement capable of this, is gamma glutamylcysteine (GGC, Glyteine), which is the immediate precursor for cellular glutathione synthesis

doi: 10.1016/S1474-4422(14)70065-1 58 FitzN

Edis Z, Wang J, Waqas MK, Ijaz M, Ijaz M

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Cell Physiol Biochem 2015