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Overall, these data suggest that NR4a1 overexpression alone does not exacerbate TAC-induced pathological cardiac remodeling but counteracts the therapeutic effect of Sema, further confirming that NR4a1 is a target of Sema in pressure overload-induced cardiac remodeling and dysfunction

In the official minutes of the board's October 14, 2010 meeting, it says: "In response to numerous inquiries and comments regarding the guidelines adopted by the CKCSC-USA, David Frederick clarified that guidelines are not medical protocols