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BPC-157 acts through multiple mechanisms, including VEGF-dependent and independent nitric oxide production, FAK-paxillin pathway activation, and gene expression modulation, without a single defined receptor

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Int (2018) 2018:16

This study suggests that knocking-out FASN in the DNL pathway affects the crosstalk between adipose tissue and nervous system

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Mechanism in research literature The most-cited mechanistic finding is that BPC-157 activates the vascular endothelial growth factor receptor 2 (VEGFR2) in vascular endothelial cells (PMID 27847966)