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An important mechanism in GIO pathogenesis is associated with increased osteclastogenesis resulting from activation of macrophage colony-stimulating factor (M-CSF) and receptor activator of nuclear factor-kappa B (NF-B) ligand (RANKL) [29]

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1b) content limits the formation of GSH, most current researches have been focused on increasing Cys levels in the brain as an indirect way to increase GSH levels 3

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These findings are based on animal models and in vitro research and have not been conclusively demonstrated in human clinical studies

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