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Marshall KR, et al

In this review article, we describe the common metabolic profile that is shared between all of these high-risk groups and COVID-19

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Some older studies have evaluated NAD+ at high doses (1-2g daily) for conditions such as chronic schizophrenia, but no improvement was noted in these studies.3 This may be because NAD+ is expected to have poor oral bioavailability.12 For this reason, when trying to boost NAD+ levels using an oral route, precursors such as nicotinamide or nicotinamide mononucleotide are typically used instead of NAD+

Animal data showed dramatic effects on synaptic connectivity, but no published human safety or efficacy trials exist as of 2026

Impaired hippocampal-dependent memory and reduced parvalbumin-positive interneurons in a ketamine mouse model of schizophrenia