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In chronically stimulated T-cells ( e.g ., in the tumor or persistent antigen context), researchers found a progressive loss of PGC-1 and other metabolic regulators, resulting in small, fragmented, and dysfunctional mitochondria in exhausted T-cells (68)

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Abecma reprograms a patients T cells to eliminate cells that express B cell maturation antigen (BCMA), which is present in normal B cells but overexpressed in multiple myeloma

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[7] Population pharmacokinetic modeling reveals a biphasic disposition (distribution half-life 15-30 minutes