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Research demonstrates that TB-500 forms functional complexes with PINCH-1 and integrin-linked kinase (ILK) that can inhibit TNF--induced NF-B activation through blocking RelA/p65 translocation, providing rapid suppression of inflammatory transcriptional programs

Previously, a Phase 3 clinical trial for the amustaline/glutathione (GSH) PR system was halted when antibodies specific to acridine (a by-product of amustaline) were found in patients

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Funding: The research described in this manuscript was supported by grant 108-1083570-3635, Ministry of Science, Education and Sports, Republic of Croatia