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Previous studies from our group demonstrated that administration of Rot, a mitochondrial complex I (CI) inhibitor, during early postnatal development (postnatal days 5 to 11) resulted in an animal model exhibiting schizophrenia (SCZ)-like phenotypes, along with alterations in mitochondrial biogenesis and synaptic protein expression 26,27

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(2021) 12:783806 doi:10.3389/fimmu.2021.783806

It was observed to be safe and well-tolerated, particularly at certain doses, with evidence of a cumulative effect after multiple doses