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The authors described these structural models as the basis for rational development of p53 inhibitors targeting the senescent cell biology field and diseases linked to cellular senescence including cancer
The resultant oxidative damage triggers an inflammatory response characterized by the upregulation of acute-phase proteins, such as CP, further exacerbating cardiac dysfunction [204]
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Beneficial commensals are depleted, while opportunistic species expand, altering local immune surveillance and perpetuating chronic inflammation
Clinical epigenetics
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