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GSH, as a central redox buffer, could influence antibiotic efficacy

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FIGURE 3 The immunopharmacological effects of GSH are evident through several distinct but interconnected pathways, including regulation of inflammatory signaling networks like MAPK and NF-B, buffering of pathologic ROS as well as maintenance of cellular ROS signaling, and modulation of both peripheral and central immune cell activity (Figure 4)

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When incorporating Glutathione-SR into a chronic illness management plan, consistency and timing are key factors for success