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reported that GLS-driven Gln metabolism is a regulator of radiotherapy tolerance in prostate cancer (PCa) and that high expression of GLS 1 and c-MYC, key regulators of Gln, are significantly associated with reduced progression-free survival in prostate cancer patients treated with radiotherapy

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When cells are deprived of oxygen, they are forced to abandon efficient mitochondrial ATP production and rely on a primitive, inefficient process called anaerobic glycolysis

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