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In rodent models of epilepsy, GLPs confer neuroprotection primarily by preventing pathological intracellular calcium accumulation and normalizing the expression levels of calcium/calmodulin-dependent protein kinase II alpha (CaMKII) (Jiang et al., 2024

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GHK-Cu, discovered earlier, is a naturally occurring tripeptide in human plasma, while AHK-Cu is a synthetic analog developed more recently

In a cohort of ME/CFS patients, the degree of mitochondrial dysfunction in neutrophils was strongly correlated with the severity of the illness (187)

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