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Rat and gerbil studies have demonstrated that intraperitoneal NAC administration protects against oxidative damage induced by acrolein, peroxynitrite, hydroxyl radicals and 3-nitropropionic acid while increasing GSH levels in the brain and synaptosomes (119123)

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We therefore hypothesized that PBAT-MPs activate a gut-to-brain inflammatory cascade that induces a lipotoxic microglial phenotype, thereby promoting neurodegeneration