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84,85 Upon activation, the C-terminal kinase domain of ATM undergoes autophosphorylation, attracting DNA repair proteins such as p53 and CHK2 to regulate the cell cycle

This includes the peptide and CTR (including the ECD position) that adopt the same conformation to those observed in Cagri-bound AMYR-Gs complexes, assessed by overlaying the backbones of the structures (Fig

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Life, death, and autophagy in cancer: NF-B turns up everywhere