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The emerging scientific consensus, highlighted by advanced research into oxidative stress and mitochondrial failure, validates what patients have known all along: your cells are fighting a massive, unseen battle at the biochemical level

If left unchecked, lipid peroxidation destroys the structural integrity of the mitochondria and triggers a highly inflammatory form of programmed cell death known as ferroptosis, a mechanism heavily implicated in the pathophysiology of ME/CFS

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On the one hand, TME immunomodulation can trigger macrophage polarization from alternately activated macrophages M2 to classically activated macrophages M1, offering intertumoral H 2 O 2 for the Fenton reaction (Zanganeh et al., 2016), which effectively generates ROS and triggers lipid peroxidation (Yang and Stockwell, 2016