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GLP-1 therapies are positioned as long-term treatments that modify biological drivers of weight, not short-term aids for dieting

Am J Respir Crit Care Med 1994;150(5 Pt 1):141622.ArticlePubMed 24

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The observation that in periventricular heterotopia FLNA null mutations caused impaired neuronal migration within the cerebral cortex (73), together with experimental evidences obtained in FLNA-deficient M 2 cells that are not able to migrate (91), suggest that FLNA is crucial in promoting cell migration

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While therian mammal GCG genes encode identical GLP-1 protein sequences, there are significant changes in the platypus Gcg ortholog