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Efficacy and safety of glucagon-like peptide-1 receptor agonists in nonalcoholic fatty liver disease: a systematic review and metaanalysis

They do this by binding to receptors in the pancreas, brain, and gastrointestinal tract, disrupting natural hunger signaling by increasing insulin release, inhibiting glucagon, and delaying stomach emptying

Combined, this multi-target action allows retatrutide to operate on various aspects of the metabolic system simultaneously

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The fat being released by tesamorelin is more effectively burned for energy because MOTS-c has improved the metabolic machinery responsible for fat oxidation