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As described in the Introduction, previous studies with little or no antibody specificity control experiments reported GLP-1R immunoreactivity primarily in glial cells, and to a lesser degree in neurons 24,25,26,27,28

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Patients with comorbid T2D and chronic lower respiratory disease on GLP-1RA had lower risk of hospitalization for exacerbation of pulmonary disease compared to those on DPP-IV inhibitors