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Additionally, ischemia-reperfusion triggers upregulation of RIPK3, promoting phosphorylation of FUNDC1 and inducing apoptosis through FUNDC1-mediated mitophagy dysfunction [113]

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PMID: 19819949

NB, like other malignancies, is associated with modifications in cellular metabolism that may contribute to oncogenesis

Nec-1 functions through the specifically targeting of Receptor-Interacting Protein Kinase 1 (RIPK1), inducing disruption in the assembly of the necrosome, consequently impeding the execution of necroptotic cell death

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