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Excessive mtROS production or impaired antioxidant defenses disrupts redox homeostasis and mitochondrial dynamics [87], leading to mtDNA damage and impaired mitophagy, thereby amplifying mitochondrial dysfunction [88, 89]

This reduction compromises the anti-inflammatory defenses of the intestinal mucosa, weakens epithelial barrier integrity, and increases oxidative stress, thereby exacerbating IBD pathogenesis [110,111,112]

Leucine supplementation attenuates macrophage foam-cell formation: studies in humans, mice, and cultured macrophages

AKT seems to play a central role as its phosphorylation appears to be severely affected in several models of both type 1 diabetes mellitus (T1DM

[DOI] [PubMed] [Google Scholar] 24.Jang A, Jo C, Kang KS, Lee M